Objective: To assess knowledge and skills level regarding oxygen administration methods at p
ediatric teaching hospitals in Mosul City.
Methodology: A descriptive study was applied at pediatric teaching hospitals (Al-Kansaa, and Ibn Al-Atheer) in Mosul City from 8 of October / 2018 till 29 of May / 2019. The selection of the sample was non- probability (Purposive). This sample involved of (52) nurses. The questionnaire was constructed which consists of three parts and provided for nurses. The questionnaire validity was carried out through a panel of experts. To evaluate statistically the reliability of instruments, the pilot study was applied through period from 20– till –31 of January / 2019. Non-randomly (6) nurses from Ibn Sina teaching hospitals, the correlation of Pearson's coefficient result are (r= 0.798) and are significant at p ≤ 0.05 level was used to approximation the scale (test – retest) by using SPSS version 25.
The Result show statistical knowledge and skills results for nurse's in concerning the oxygen administration methods for pediatric, that knowledge results are 84.6% (44) of them at not acceptable level, and the skills results are 65.4 % (34) of them also at not acceptable level. There are not significant relationships between the knowledge, skills results and all the demographic variables except the skills results with training courses only there are significant relationships at p≤ 0.05.
Recommendations: Training course and workshops for nurses of teaching hospitals regarding oxygen administration methods
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Escherichia coli (E. coli) is a frequent gram-negative bacterium that causes nosocomial infections, affecting more than 100 million patients annually worldwide. Bacterial lipopolysaccharide (LPS) from E. coli binds to toll-like receptor 4 (TLR4) and its co-receptor’s cluster of differentiation protein 14 (CD14) and myeloid differentiation factor 2 (MD2), collectively known as the LPS receptor complex. LPCAT2 participates in lipid-raft assembly by phospholipid remodelling. Previous research has proven that LPCAT2 co-localises in lipid rafts with TLR4 and regulates macrophage inflammatory response. However, no published evidence exists of the influence of LPCAT2 on the gene expression of the LPS receptor complex induced by smooth or rough b
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